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Administration ofthe ROS inhibitor N- acetylcysteine (NAC) partially restored GSH, attenuated ROS accumulation, and modestly alleviated Gln-deprivation-induced growth inhibition (Fig

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Clinically, strategies aimed at modulating EAAC1-mediated cysteine transport and boosting endogenous GSH synthesis may hold promise for mitigating post-ischemic neurodegeneration and cognitive deficits associated with vascular dementia