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I n recent years, cancer-causing compounds have been identified in other common drugs, and the FDA has been working with international regulators to address and mitigate drug contamination issues

It is proposed that the non-ubiquitinated NRF2 would remain bound to the KEAP1-Cul3 complex and therefore prevent the newly translated NRF2 from inhibition by the E3-ubiquitin complex, thereby promoting NRF2 accumulation in the nucleus [17]

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