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Considering the physiological role of selenoproteins in antioxidation and the detrimental effects of oxidative stress in chondrocytes, aberrant selenium metabolism is likely to disrupt cartilage homeostasis and cause arthropathic diseases via dysregulation of redox homeostasis (Fig

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Curr Pharm Biotechnol 12(8):11011116 Thevenard J et al (2006) Structural and antitumor properties of the YSNSG cyclopeptide derived from tumstatin