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As has been mentioned previously, this increased GPx activity, most probably, is a compensatory response to increased lipid peroxidation occurring in riboflavin deficiency

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Moreover, the presence of NAFLD is a robust predictor of MetS [68]

An important factor in this polarization is Nrf2 activity: strong Nrf2 signaling inhibits NF-B-mediated NLRP3 priming, boosts antioxidant defenses via HO-1, NQO1, and glutathione formation, and encourages a change to an M2-like phenotype that aids in tissue regeneration [62]