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This chelation reaction can occur in both intra- and extra-cellular environments due to the lipophilic properties of dimercaprol, but it is most prominent within extracellular compartments

CzEcp2, CzNIS1a and CzNIS1b were subsequently transiently expressed in model plant systems and shown to trigger non-host resistance, suggesting they target conserved structures or processes to facilitate pathogen colonization and growth during the switch to necrotrophy