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Researchers hypothesize this discrepancy may be linked to lorazepams agonistic activity on GPR68

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These results indicate a close association between GPCR desensitization and the nuclear translocation of G and arrestin3

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Although the NPY modulation has been explored in obesity, alcoholism, anxiety, depression, epilepsy and pain (reviewed in [34, 182, 183]), its potential benefit in asthma is largely unknown and unexplored

In addition to these major sources, the endoplasmic reticulum can contribute to the ROS burden (Zhang et al., 2025)