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Figure 2 4.3.2 Amplification of immune responses by released intracellular components following ferroptosis When ferroptosis leads to the rupture of chondrocytes or synovial cells, the oxidized lipid byproducts, iron ions, and damage-associated molecular patterns (DAMPs) that leak from these cells are detected by nearby immune cells

Notably, there is an overlap in cellular and molecular alterations of lamin A protein in both PD and Hutchinson-Gilford Progeria syndrome which develops due to a deficiency of lamin A protein (Al-Kuraishy et al

In comparison to the docking conformation, the chelation site of CuB shifted from the ether oxygen to the hydroxymethyl group, with the hydroxyphenyl group forming a hydrogen bond with S356

Youve introduced a potential entry point for contaminants