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In U87 cells, treatment with AKT inhibitor (HY-10249D) resulted in downregulation of GLUT1 and GLUT6 expression, while SREBP2 expression was downregulated (Fig

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10.1056/NEJM199007053230101 79 SathiavageeswaranM.BurmanP.LawrenceD.HarrisA

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extended this finding by revealing that phosphorylation modifications of liver-derived Fetuin-A in human serum and CSF are linked to CNS inflammation in AD, suggesting that post-translational regulation may further impair its functionality under pathological conditions [7]