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Plates were read at 405 nm and titers were defined as the log 3 serum dilution two-fold above background

We and others have demonstrated the biochemical and molecular mechanisms of alcohol-induced oxidative production as contributing factors to neuronal degeneration after alcohol-induced breaching of brain microvascular endothelial cells [15,16], brain astrocytes [17,18], and cultured microglia [19]

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DNA nanoflower Oligo-PROTAC for targeted degradation of FUS to treat neurodegenerative diseases

In silico prediction of the effects of nonsynonymous single nucleotide polymorphisms in the human catechol-O-methyltransferase (COMT) gene