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Effects of amygdalin on ferroptosis and oxidative stress in diabetic retinopathy progression via the NRF2/ARE signaling pathway

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Remarkably, this inflammation-targeting SP@M restored osteoimmunology homeostasis, inhibited the activation of osteoclasts, protected bone formation, and promoted cartilage recovery in CIA mouse models in vivo (Fig

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First, we assessed the viability of the MNA IMR-5/75 cells in response to MitoQ