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Kataria, R

Of significance to diabetes is that ER stress-induced inflammation activates the kinases JNK (JUN N-terminal kinase) and IKK (inhibitor of nuclear factor-kappa B kinase subunit beta) that phosphorylate insulin receptor substrates (e.g

Assessment of glutamate transporter GLAST (EAAT1)-deficient mice for phenotypes relevant to the negative and executive/cognitive symptoms of schizophrenia