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It is reactive VPA metabolites that are the substances that cause damage to biosystems at the molecular level [30]

When mitochondrial depolarization damage occurs, the mitochondrial membrane potential (MMP) decreases, and PINK1 hydrolysis is inhibited, which stably binds to the damaged mitochondrial outer membrane, recruiting Parkin to translocate from the cytoplasm to the mitochondrial outer membrane, thereby phosphorylating and activating Parkin activity (Uoselis et al., 2023)

The OH depolymerizes GI mucin, inactivates pyruvate dehydrogenase, an essential mitochondrial enzyme, and damages mitochondrial RNA and DNA in the GI tract (113)

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