Glutathione helps maintain an environment where these lipids can function effectively without being damaged by oxidative stress
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doi: 10.3389/fphar.2024.1358573 Received 20 December 2023 Accepted 11 March 2024 Published 27 March 2024 Volume 15 - 2024 Edited by Divya Bhatia, Weill Cornell Medical Center, NewYork-Presbyterian, United States Reviewed by Richard Pratley, AdventHealth, United States Oscar Lorenzo, Health Research Institute Foundation Jimenez Diaz (IIS-FJD), Spain Keiichiro Matoba, Jikei University School of Medicine, Japan Updates Copyright 2024 Muta, Kobayashi, Toyoda, Tone, Suzuki, Tsuriya, Machimura, Shimura, Takeda, Yokomizo, Takeshita, Chin, Kanasaki, Tamura, Miyauchi, Saburi, Morita, Yomota, Kimura, Hatori, Nakajima, Ito, Tsukamoto, Murata, Matsushita, Furuki, Hashimoto, Umezono, Takashi and Kawanami
MitoTAD clears precursor proteins blocked in the TOM channel, and it depends on the key molecules involved in ERAD and Cdc48 (also known as AAA-ATPase p97) (Fig
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Endothelial dysfunction is a significant component of HFpEF pathophysiology, and glucagon-like peptide-1 receptor (GLP-1R) agonists have shown potential benefits in improving endothelial function
