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10.1002/hbm.23085 Hum

223 Hence, related to hypoxia and ER stress genes could be considered as precise therapeutics to control the IR

Boogaard, P.J., Zoeteweij, J.P., Van Berkel, T.J.C., Vant Noordende, J.M., Mulder, G.J

tirzepatide, about 20%

Such modifications then activate NF-B that is involved in the expression of proinflammatory mediators such as TNF and IL-6, 343,344 and that induces key steroidogenic molecules, like CYP11A1, CYP17A1 and StAR, leading to further aggravation of hyperandrogenemia

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