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When administered as a once-weekly subcutaneous injection, semaglutide stimulates insulin secretion from pancreatic beta cells in a glucose-dependent mannermeaning it only promotes insulin release when blood glucose levels are elevated

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This effect may be associated with the restoration of glutamatergic neuron populations within the BLA region of the brain, subsequently resulting in a reduction in the projection of the neural repair protein GLP-1 (Fig 7)

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This adaptation, called receptor downregulation, typically occurs between months 3 and 8