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doi: 10.1164/rccm.202010-4017ED

J Insur Med

In long-term evolution, human UA kinase factors and a series of promoter gene mutations have caused humans to have higher UA levels than other mammals (Figure 1) (Edwards, 2009)

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The end product of this reaction (AGE), in turn, induce oxidative stress and accelerate the Maillard reactions ultimately leading to inflammation and the propagation of tissue damage [30, 31, 32]