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By increasing availability of substrate for acetylation and shifting the T cell epigenome, LDH promotes Th1 commitment and IFN- production (64)

demonstrated that exogenous IGF-1 administration reduces functional deficits and accelerates recovery in a rat Achilles tendon injury model ( In vitro studies further support its therapeutic potential, showing that IGF-1 modulates inflammatory responses, stimulates tenocyte proliferation and migration, and enhances collagen synthesis (Olesen et al., 2021)

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