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We now demonstrate that GIPR agonist administration critically relies on Gipr expression in the hindbrain, while GIPR antagonism relies on hypothalamic Gipr expressionboth are probably neuronal populations, given that we used hSyn-driven Cre-expression for the region-specific knockout studies

Increasing the activation of antioxidant genes: Nrf2 regulates antioxidant and detoxification genes and can protect against oxidative stress in chronic diseases [4]

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