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Lasisi AO, Fehintola FA

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Journal of Molecular Medicine (2017) Research demonstrating enhanced angiogenesis with BPC-157 in ischemic rat hind-limb muscle and vascular endothelial cells, via increased expression and internalization of VEGFR2 (but not VEGF-A).

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The rationale is mechanistic non-overlap: rather than two compounds acting through the same pathway with redundant effects, BPC-157 and TB-500 engage distinct targets the former operating through nitric-oxide-pathway and VEGFR2 modulation, the latter through actin-cytoskeleton regulation

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