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Similarly, NOX4 has been shown to be overexpressed in various tumor types and implicated in cellular proliferation and survival [35]

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Finally, integration of in vitro scratch-wound assays under basal, inflammatory (LPS), and oxidative (H 2 O 2 ) stress conditions provides mechanistic corroboration that HA + GSH-C4 may exert a direct pro-repair effect on corneal epithelial cells