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was supported by the Alexander von Humboldt fellowship

G6PD deficiency is an X-linked disorder and thus deficient males and homozygous deficient women will exhibit the full extent of the deficiency whereas G6PD heterozygous women will have a range of G6PD activities from deficient to very normal G6PD activity

Fe 2+ catalyzes, in the presence of hydrogen peroxide, the formation of the hydroxyl radical OH by means of the Fenton reaction, while the reaction of Cu + with H 2 O 2 leads to the formation of OH and O 2 radicals [86]

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Moreover, there was a mild increase in Ucp1 expression (Extended Data Fig

The simultaneous exposure of rabbits to citrinin with OTA even at sub-clinical dietary levels potentiated the OTA induced nephrotoxicity at ultrastructural level [146]