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None of the clinical trial participants at 1mg/day experienced serious adverse events

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TNF- damages cardiomyocyte mitochondria by inhibiting glycolysis and reducing mitochondrial adenosine triphosphate (ATP) production through promoting oxidative and nitrosative stress, which can result in compromised cardiac systolic function and unfavorable cardiac remodeling [83, 84]

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IGF-1 in the Upper Half of Normal Range: Generally indicates adequate growth hormone production