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[] SFN is excreted by forming a glutathione conjugate via the enzyme glutathione-S-transferase M1 (GSTM1), [] but half of the population does not express GSTM1 either because of polymorphism or homozygous loss of the gene

Rasmussen J, Nielsen OW, Janzen N, et al

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These heterodimeric receptor assemblies generate multiple receptor subtypes, commonly referred to as AMY1, AMY2, and AMY3 receptors