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Both work by targeting specific receptors involved in appetite regulation, glucose metabolism, and energy balance, but they do so through distinct hormonal pathways

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On the other hand, studies in a model of induced diabetes in rats showed a higher concentration of H 2 S in the liver and pancreas compared with healthy control, which was accompanied by an increase in the expression of enzymes responsible for H 2 S synthesis in these organs [127,128]

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An invention was submitted for a patent on 12 November 1996 [7], and the scientific article was submitted for publication in the Journal of Clinical Investigation 20 June 1997 and published 1st February 1998 [6]