GHK (in this experiment the peptide alone, not its copper complex, was used) has been proven to quench alpha,beta-4-hydroxy-trans-2-nonenala toxic product of fatty acids' lipid peroxidation that play important role in the pathogenesis of several age-related conditions including Alzheimer's disease, neuropathy, and retinopathy [39]
Higgins, L.G., et al., Transcription factor Nrf2 mediates an adaptive response to sulforaphane that protects fibroblasts in vitro against the cytotoxic effects of electrophiles, peroxides and redox-cycling agents
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