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used exendin-4, an exendin-based GLP-1 analog, in hyperglycemia-induced bronchial hyperresponsiveness ex vivo, which resulted in bronchodilation that was inhibited by either cAMP-PKA antagonism or GLP-1 RA [90], a result consistent with other findings (Fig

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The concordance between the proteomic profile and the alterations observed in oxygen consumption, mitochondrial membrane potential, mitochondrial morphology, cellular redox balance, proliferation, migration, and apoptosis provides a coherent framework for unravelling the biological effects of this compound

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