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These perceptions about the changes in receptors, thyroid hormone metabolism, and synthesis have been augmented by research involving transgenic mouse models that have thyroid hormone receptors and TSH receptor mutations

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Contributing factors include: reduced nutritional intake (proteins, essential amino acids, competition between amino acid transporters, vitamin insufficiency [riboflavin: GSSG reductase, nicotinic acid: NADPH, pyridoxine (B 6 ): SAMe pathway], mitochondrial dysfunction: ATP deficiency, down regulation SAMe synthase, oxidation of SAMe pathway enzymes, enhanced GSH utilization: conjugation reactions (toxins, drugs), and overwhelming oxidative challenge

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The answer is a classic, and often frustrating, 'it depends.' Its not a simple yes or no