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Glycated albumin increases oxidative stress, activates NF-kappa B and extracellular signal-regulated kinase (ERK), and stimulates ERK-dependent transforming growth factor-beta 1 production in macrophage RAW cells

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However, recent research challenges this simplistic view by revealing that excessive or prolonged antioxidant supplementation can shift the cellular redox environment toward RS a state marked by an overabundance of reducing equivalents, such as glutathione and NADPH [8, 9]

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