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Selective effects of kainic acid on diencephalic neurons

[DOI] [PMC free article] [PubMed] [Google Scholar] Kauffman FC (2004) Sulfonation in pharmacology and toxicology

It is synthesized from three amino acids in two ATP-dependent steps, beginning with the formation of a peptide bond between -glutamate and cysteine by -glutamylcysteine synthetase (GSH1) and the subsequent addition of glycine, catalyzed by glutathione synthetase (GSH2) (Figure 1: GSH biosynthesis) [20]

Neurotoxicology 78, 163169

These compounds are being explored for their ability to not only mitigate oxidative damage but also to modulate bone metabolism and offer effective therapeutic strategies for managing osteoporosis and related bone disorders

In AD patients, lipid peroxidation plays an ambient factor in neuronal death through various mechanisms, including the inappropriate function of ion channels (Na + , K + , and Ca ++ ) for glucose transporters (Fig