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[246] showed that Nrf2 inhibition increased oxidative stress and aggravated hypertension in mice, whereas Nrf2 activation alleviated hypertension during Ang II infusion, highlighting its therapeutic relevance.Mitochondrial dysfunction, including impaired energy metabolism, increased mtROS, and mtDNA damage, amplifies ferroptosis and worsens endothelial injury [247, 248]

Subjects for the study were identified from the glaucoma clinics at the Fondazione Policlinico Tor Vergata

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Treatment of macrophages with 25HC triggered FAK phosphorylation as detected by western blot analyses (Fig