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In traumatic brain injury mice, Cb supplementation reduces inflammatory reactions and intestinal permeability, thereby improving the neurological dysfunction and BBB injury, likely due to increased GLP-1 secretion (154)

Participants were instructed to pause daily doses of short-acting GLP1-RAs on the day of surgery and continue long-acting GLP1-RAs before surgery according to established clinical practice

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However, the triagonist failed to enhance insulin secretion in islets lacking both Glp-1r and Gipr and upon treatment with the GLP-1 receptor-specific antagonist exendin-3 (939)