Semaglutide reduced systolic blood pressure by 3.3 mm Hg, high-sensitivity C-reactive protein by 37.8%, and improved lipid profilesall achieved on top of high baseline rates of evidence-based cardiovascular therapies (90% on statins, 86% on antiplatelet agents).[1] These pleiotropic effects likely contribute to cardiovascular risk reduction through mechanisms distinct from adiposity changes, supporting the reconceptualization of GLP-1 receptor agonists as cardiovascular disease-modifying agents rather than simply weight-loss medications
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Perivascular spaces in the context of cerebral small-vessel disease Cerebral small-vessel disease commonly accompanies aging and is frequently identified on MRI through a constellation of features, including small subcortical infarcts, lacunes, white matter hyperintensities, enlarged PVS, cerebral microbleeds, and global brain atrophy
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Side effects of semaglutide typically lessen over time, but if your body doesnt adjust well to these symptoms this may also be cause for a smaller dose or discontinuing the medication altogether
10.1371/journal.pone.0112845 45 LiQ
