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This can mediate the influx of GSH through a Ca 2+ -sensitive pathway [51]

Transcriptional dysregulation in Huntington's disease: the role in pathogenesis and potency for pharmacological targeting

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Understanding these sources and metabolic pathways provides a foundational rationale for CoQ10 supplementation in clinical contexts, particularly in states of deficiencysuch as aging and ovarian dysfunctionwhere endogenous synthesis or dietary intake is insufficient to meet reproductive demands

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