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Amongst the many mechanisms postulated to contribute to its antimelanogenic properties (Figure 1), inhibition of tyrosinase enzyme, skewing of melanogenesis from the darker eumelanin to the lighter phaeomelanin, and scavenging of free radicals seem to be the most important [8]

Disruption of NOS1 dimerization can lead to abnormal CDK5 phosphorylation and activation, which impede synaptic plasticity and promote AD development [83, 84]

The ability to co-utilize glucose and xylose to reach this titer could make these cells ideal for l-DOPA production using hydrolyzed lignocellulosic biomass

DHODH-mediated ferroptosis defence is a targetable vulnerability in cancer

What is the theoretical concern about senolytics and cancer

L-carnitine supplementation in such individuals may prevent or reduce high ammonia levels ( hyperammonemia ) and the severity of liver and brain damage, according to some clinical trials [27]